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Isoflurane provides rapid administration of anesthesia and particularly rapid way out of it. Although a slight irritant effect may limit the speed of doing business in anesthesia, however, the drug does not cause increase in salivation or tracheobronchial secretions. Pharyngeal and laryngeal reflexes are quickly suppressed. The level of anesthesia with isoflurane application can change rapidly. Heart rhythm remains stable. With the deepening of anesthesia is suppressed spontaneous breathing, so it should be carefully monitored and maintained as needed.
During the induction of anesthesia reduces blood pressure trenbolone enanthate, which is quickly returning to normal after the operation.
In supporting anesthesia blood pressure is reduced in proportion to the depth of anesthesia, but cardiac rhythm It remains stable. With controlled respiration and normal , cardiac output usually remains unchanged despite the deepening of anesthesia due to an increase in heart rate which compensates for the reduction in stroke volume. With spontaneous breathing develops hypercapnia can lead to increased heart rate and cardiac output, which exceed those of waking up.
Cerebral blood flow is not altered by light isoflurane anesthesia but tends to increase in its recess. To prevent elevation of cerebrospinal fluid pressure or reduce hyperventilation is recommended before or during anesthesia.
Electroencephalographic changes and convulsions when applying isoflurane observed only rarely. In general isoflurane causes  changes comparable to those when using other inhalation anesthetics.
Isoflurane increases sensitivity to adrenalin myocardial even less than enflurane. There is limited evidence that subcutaneous infiltration of up to 50 ml solution of epinephrine trenbolone enanthate does not cause ventricular arrhythmias in patients who are under isoflurane anesthesia.
At normal levels of anesthesia may be sufficient muscle relaxation for some intra-abdominal operations. If you want a more pronounced muscle relaxation may intravenous administration of small doses of muscle relaxants.
Isoflurane may be used for induction of anesthesia, and maintenance of anesthesia.
The possibility of its use in pregnancy has not been established adequate studies.

Isoflurane undergoes relatively little metabolism in humans. Postoperatively as metabolites detected in urine dose only 0.17% isoflurane. Peak serum concentration of inorganic fluoride is typically less than 5 mol / l and observed after 4 hours after anesthesia. Fluoride level returns to normal within 24 hours. It was registered signs of kidney damage after using isoflurane.
Known metabolites of isoflurane did not have toxicity or determined in too low concentrations.

Background and supporting general anesthesia.
The drug can be used for sedation trenbolone enanthate for 48 hours in patients undergoing mechanical ventilation (ALV) in the intensive care unit.

Hypersensitivity to isoflurane and malignant hyperthermia in history.